From the studies reviewed above, it seems that proteinuria is not a consistent feature of ovine pregnancy toxaemia.
It is quite possible that with prolongation of the uteroplacental ischaemia, proteinuria and renal pathological changes would develop (see below).
The presence of gestational proteinuria may no doubt contribute to false positive referrals to obstetric day units and potentially lead to iatrogenic intervention.
Crescentic glomerulonephritis should be considered in any elderly patient presenting with acute renal failure associated with haematuria, red cell casts and proteinuria.
Thus, proteinuria may have been overlooked in many animals with compromised uteroplacental perfusion and increased arterial pressure.
Investigation revealed hypoproteinemia with depressed concentrations of albumin in the serum, normal kidney and liver function, and no proteinuria over a period of 24 hours.
A hypothesis proposing increased blood viscosity as a cause of proteinuria and increased vascular permeability.
It is possible that there is a microalbuminuric phase that precedes overt proteinuria in preeclampsia and that we might devise strategies to detect this.
