0 the collection of amyloid in the tissues or organs of the body
Familial atypical generalized amyloidosis with special involvement of the peripheral nerves.
Death can result from infections, haemophagocytosis, renal or cardiac amyloidosis, or myocardial infarction.
A chronic abnormality of the immune system is the mechanism frequently proposed for the pathogenesis of the amyloidosis.
Vitreous amyloidosis associated with homozygosity for the transthyretin methionine-30 gene.
A role for destabilizing amino acid replacements in light chain amyloidosis.
However, even during symptom-free intervals, these patients can be shown to have an intermediate degree of biological inflammation, which increases the risk of amyloidosis.
Secondary autonomic failure may be associated with diabetes, chronic renal and hepatic failure, connective tissue disorders, amyloidosis, demyelinating polyneuropathies, multiple sclerosis and brainstem lesions.
Meningocerebrovascular amyloidosis associated with a novel transthyretin mis-sense mutation at codon-18.
