0 the action of making blood flow return to normal after the supply has been cut off, especially after a heart attack or stroke: --
Rewarming is commenced, and, during reperfusion, pacemaker wires are placed on the atrium and ventricle, and a pulmonary venous catheter is inserted.
Following cerebral ischemia and reperfusion there is a downregulation of the protein synthesis machinery and translation arrest.
Taken together, these findings illustrate that the brain has a heterogeneous response to ischemia and reperfusion injury.
Compared to sham controls, myelin processes began to degenerate at 12 hours reperfusion, with a significant loss of intact processes by 48 hours.
Addition of insulin to those solutions may be beneficial to protect immature myocardium from hypothermic, ischemic or reperfusion injury.
Our findings of differential regional reperfusion following hypothermic circulatory arrest also have important implications.
Alternatively, excess formation of oxygen radicals after arrest associated with reperfusion may cause or exacerbate cerebral injury.
With the resumption of blood flow, the subsequent reperfusion produces large quantities of superoxide radical and hydrogen peroxide.
