Focal psychological syndromes such as aphasia and ataxia may herald the onset of the disease.
The spectrum of genetic ataxias continues to expand.
This syndrome combines gait ataxia and a predominantly sensory neuropathy, with severe functional disability.
This upregulation is associated with the late-onset ataxia observed in these mice.
According to the planning-control model, a patient suffering from optic ataxia has deficits that are limited to on-line control but has unimpaired movement planning.
These symptoms include dysarthria, vertigo, tinnitus, ataxia, bilateral paresis or paresthesia.
Symptoms commonly include monocular visual loss, vertigo, diplopia, weakness, numbness and ataxia.
The gross neurologic side-effects include ataxia, dysarthria, nystagmus, dizziness, unsteadiness, blurring and doubling of vision, reversible dyskinesias and asterixis.
