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This loss of muscular coordination is called ataxia.
Multisystem atrophy is generally referred to as ataxia or the loss of the ability to coordinate muscular movement.
Historical accounts about President Lincoln and his sometimes awkward gait suggest he may have had ataxia.
This contrasts sharply with the apparent contralateral organization of parietal mechanisms involved in on-line control that are damaged in optic ataxia.
Focal neurological signs include upper motor neurone weakness, lower cranial nerve palsies, tremor and ataxia.
Because of his ataxia, he can shop and cook only with great difficulty.
Patients with these disorders display profound oculomotor abnormalities and ataxia.
On examination, typically in addition to downbeat nystagmus, lower cranial nerve palsies and both gait and limb ataxia are noted.
If a specific cause for the ataxia can be identified and treated, such as a paraneoplastic syndrome, symptoms could stabilize after treatment.
Common side effects of carbamazepine in children and adolescents include sedation, ataxia, dizziness, blurred vision, nausea, and vomiting.
The gross neurologic side-effects include ataxia, dysarthria, nystagmus, dizziness, unsteadiness, blurring and doubling of vision, reversible dyskinesias and asterixis.
Symptoms commonly include monocular visual loss, vertigo, diplopia, weakness, numbness and ataxia.
These symptoms include dysarthria, vertigo, tinnitus, ataxia, bilateral paresis or paresthesia.
According to the planning-control model, a patient suffering from optic ataxia has deficits that are limited to on-line control but has unimpaired movement planning.
This upregulation is associated with the late-onset ataxia observed in these mice.
This syndrome combines gait ataxia and a predominantly sensory neuropathy, with severe functional disability.
The spectrum of genetic ataxias continues to expand.
Focal psychological syndromes such as aphasia and ataxia may herald the onset of the disease.
