0 the action of making blood flow return to normal after the supply has been cut off, especially after a heart attack or stroke:
Although myocardial reperfusion can rescue the number of necrotic cardiac myocytes, it is critically dependent upon the time of intervention after ischaemic insult.
The effect of oxygen radicals during reperfusion producing peroxidation and cellular injury is well described for other tissue beds during normothermia.
And finally a reperfusion injury characterized by free radical formation and oxidative stress.
This difference was particularly marked at 30 minutes, 60 minutes, and 90 minutes of reperfusion.
Other laboratory studies have supported our finding of differential regional reperfusion.
There are no additional studies, either animal or clinical, which discuss the time of reperfusion necessary to replete cerebral stores of energy to normal.
The permeability of the blood-brain barrier was highly increased by ischemia/reperfusion.
With the resumption of blood flow, the subsequent reperfusion produces large quantities of superoxide radical and hydrogen peroxide.
