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Subadult individuals were all black-coated wild boar whose carcass weighed <50 kg, supposedly corresponding to 13- to 24-month-old animals.
Indeed, a high specificity (94%) has been recorded in young wild boar.
Young individuals were all reddish-brown coated or strayed wild boar and were assumed to be aged <12 months.
During the same period 15 593 spleens and 8013 sera were analysed from hunted wild boar.
An increase in the proportion of wild boar alleles across the genome increased length versus width of femur and reduced the prevalence of osteochondrosis.
There were big populations of larger mammals such as elk, deer, bears, wolves and wild boar.
The proportion of wild boar alleles averaged over the genome was then calculated from these probabilities.
Two mechanisms may explain the decrease of incidence in young wild boar over time.
Our third hypothesis is related to the spatial structure of wild boar populations.
Each of the three infected wild boar harboured only one larva.
Finally, hunting parties may push wild boar from their natural home range as they flee from hunters.
We hypothesize that the spatial and social structure of wild boar populations may be responsible for this variability of incidence over space.
Moreover, it has been argued that the original density of the population may be quickly re-established as a result of the high fertility of wild boar.
Significance of natural biocenoses and agrocenoses as the source of food for wild boar.
However, in the present case, using a novel statistical approach, this gastrointestinal parasite was identified as an indicator species of wild boar of more than one year of age.
