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Involvement of the ophthalmic branch of the trigeminal nerve is particularly serious in that it can damage the cornea and iris.
Both procedures preferentially damage the non-myelinated fibres of the trigeminal nerve.
The left and right trigeminal ganglia from each animal were embedded together and cut longitudinally at 10 mm on a cryostat.
Drugs used in the management of trigeminal neuralgia.
The results show that excess spalt affects the localization of neural crest cells within the trigeminal ganglion.
By contrast, few (30% of the population) spalt-electroporated neural crest cells entered the trigeminal ganglion.
Dental disease is referred to the distribution of the trigeminal nerve.
Expressing the gene in neural crest cells by electroporation, inhibits migration of neural crest cells into the trigeminal ganglia, and prevents differentiation into neurons or other differentiated cells.
After over-expression by electroporation into the cranial neural tube and neural crest, we observed a marked redistribution of electroporated neural crest cells in the vicinity of the trigeminal ganglion.
These investigators found latent viral genomes in about 3% of sensory neurons in positive trigeminal ganglia and calculated that the average copy number of latent genomes per cell was 16.
We focused our studies particularly on the trigeminal ganglion, a cranial sensory ganglion that receives a contribution from neural crest cells derived from the midbrain and rostral hindbrain.
In these studies it has been shown that vocalizations can be elicited from cranial nuclei in the brainstem, such as the solitary nucleus, trigeminal motor nucleus, and the reticular formation.
The pain usually commences in the maxillary and mandibular divisions of the trigeminal nerve and in less than 5% of cases begins in the ophthalmic division.
During primary infection, virus enters sensory nerve terminals and is transported retrogradely via axons to neuronal cell bodies i n the trigeminal ganglion where latency is established (see below).
Reactivation of the virus in the trigeminal ganglion seeds to the oral or nasal mucosae, most commonly at the vermillion border of the lip, to produce characteristic vesicles.
