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It is fully explicit, quantitative, and economical, and it brings in known features of the visual system (differential magnification) and oculomotor system.
Direct connectivity between subcortical nuclei (vestibular, collicular) and the oculomotor complex exists and these structures could interact independently of the cortex.
An intuitive explanation can be given in terms of the influences of the spatial frequencies of the visual stimulus on neural activity during oculomotor behavior.
We shall also use the oculomotor-cognitive dimension to organize our discussion, starting with those models that assign the least significance to linguistic processing.
In addition, oculomotor tests were given at regular intervals, but less frequently than alertness and cognitive performance tests.
However, when considered in the context of differences in the oculomotor habits of these two animals, we believe the homology fits.
In addition, labeled somatic motoneurons (diamonds) are present in and ventrolateral to the oculomotor nucleus.
They selectively labeled the smaller neurons located ventromedial to the oculomotor nucleus, but produced little or no labeling within the boundaries of the nucleus.
Oculomotor programming is influenced by the information in the word that is the target of the next saccade.
However, for the appropriate oculomotor neuron, a tight linkage between its activity and movement of the eye is expected.
Specifically, they are found in and lateral to the anteromedian nucleus, and they extend caudally, both dorsal and ventral to the oculomotor nucleus.
For most neurons, position of the fixation point (and, hence, oculomotor position) did not affect activity.
The oculomotor system has access only to the output of the motion-processing stage, which it uses to generate pursuit.
The final group of cells was found ventral to the oculomotor nucleus and medial longitudinal fasciculus.
Painful oculomotor paresis with retro-orbital pain is usually due to one of two main pathologies.
