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In the normal small intestine, intact intercellular tight junctions act as a barrier to antigens such as gluten.
Similar results were found for dry gluten (data not shown).
Non-compliance with regard to the gluten-free diet is the most common cause of failure to respond to diet.
This reflects the interaction between genetic predisposition and ingestion of gluten-containing grains necessary to precipitate the disease.
It is only recently that the deleterious part of gluten has been identified and a method developed for the measurement of gluten-antibodies in the blood.
The main source of gluten is wheat, but the prolamins in rye and barley are also toxic and must be avoided.
A rechallenge with gluten is perhaps unnecessary in the elderly.
Compliance is aided by the frequent rapid symptomatic response to gluten withdrawal.
In addition, protein and gluten concentration of grains were measured.
Although the rash and intestinal pathology are shown to be gluten-dependent, it is not known how gluten produces the lesion.
There is no evidence as yet that avoidance of gluten reduces the risk of lymphoma.
The pathogenetic significance of gluten is unresolved.
Repeat biopsy should be performed if there is diagnostic doubt or if the clinical response to a gluten-free diet is inadequate.
Wheat flour containing gluten is present in a wide variety of processed foods and even in some medication formulations.
Dough properties and baking performance of wheat are determined by the structure and quantity of gluten proteins and are strongly dependent on both the genotype and the growing conditions.
