Príklady carcinogenesis

• As further novel genes and markers are identified in human prostate carcinogenesis, it is essential that we base new murine models on these findings.

• Understanding the function and interactions of the family as a whole, in both cell growth control and carcinogenesis, will be important.

• The passage of time is a ubiquitous carcinogen to which we are all exposed but age itself is not the mechanism of carcinogenesis.

• Low folate status has been implicated in carcinogenesis, possibly as a result of a higher rate of genetic damage.

• Interactions of alcohol with host and environmental factors, alcohol-related various procarcinogens, inherited susceptibility, and methylation have also been involved in the breast carcinogenesis.

• Several studies have revealed variations in its cell differentiation and carcinogenesis functions.

• Carcinogenesis can result either from the expression of a mutant version or from an altered gene product.

• Thus, the search for and identification of these checkpoint mediators naturally led to the recognition of their potential roles in mediating carcinogenesis.

• Although mice do not spontaneously develop prostate cancer, the murine models of human cancer have provided new tools for the investigation of prostate carcinogenesis.

• Consistent evidence from animal experiments shows that ethanol modulates chemically induced carcinogenesis.

• Problems such as fistulae, fibrosis, tissue necrosis and carcinogenesis may therefore arise in the future.

• Mutation of a stem cell is not the only mechanism for carcinogenesis.

• What is involved in testing a material for carcinogenesis?

• Secondly, mutagens are always high on the suspect list for carcinogenesis.

• In the long term there are some worries about paranoia, memory loss and carcinogenesis.