However, we find that rat ganglion cell spike trains are reasonably stationary for certain anesthetics but not for others commonly used in vision experiments.
These data indicate that anesthetic action in the spinal cord indirectly suppresses ascending transmission of somatosensory information, thereby altering the 'arousal' level of the brain.
As suggested above, the indirect spinal action of anesthetics might 'reset' the arousal level of the brain by reducing the excitability of reticulo-thalamo-cortical arousal mechanisms.
For bulk nerve labeling, fish were anesthetized, a moistened absorbent cloth placed over the body and the gills continually perfused with anesthetic solution.
The separate handling of this information is particularly pertinent within the context of economic evaluations of anesthetic techniques.
Observational studies found no difference between the anesthetic arms in terms of staff input, thus, no difference in semi-fixed costs was found.
To integrate information from such studies into a broader understanding of pain requires knowledge about how anesthetics influence sensory processing.
Rather, the local anesthetic lidocaine was topically applied to the sclera prior to injection.
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