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Initial studies on in-utero tracheal occlusion were undertaken by tracheal ligation,111 using clipping of the fetal trachea.
From top left to right bottom: epiglottis, vocal cords, trachea, carina, inflated and detached balloon, vocal cords about to close over the balloon.
All are oriented with the anterior to the right and show the mouthparts, the two dorsal branches of the tracheae and the midgut.
From that point on, the embryos that did not hatch progressed through the stages of brown midgut, brown tracheae and death.
At its upper end, the trachea enters the larynx, which in turn opens into the pharynx.
A femoral vein and the trachea were cannulated.
This nematode was found in the air sacs and trachea in nearly equal numbers (table 1).
The fetal trachea is filled with amniotic fluid, and thus readily seen using sonography.
However, we were unable to dissect the tracheae without damaging them, so we cannot exclude dissection damage as the cause of this staining.
The placement of the trachea is, however, such that food can sometimes fall into it and end up in the wrong place.
Both genotypes therefore display the same progression of phenotypes, from brown midgut to brown tracheae to death.
It then descended behind the trachea and esophagus to supply the posterior and medial segments of the right lower lobe.
This uncommon abnormality can give rise to symptoms due to compression of the trachea and esophagus.
In particular, there was no evidence of any cytopathogenic or inflammatory change in the trachea or lungs.
The trachea was canulated, and the left and right superficial subcutaneous forearm veins canulated.
