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Second, platelet counts should be followed during the pregnancy, as often as every 2+4 weeks, in order to detect thrombocytopenia as it develops.
Recent work (ours and others) with antiplatelet antibodies has raised another issue: coincidence of auto- and allo-immune thrombocytopenia.
Without conclusive statistical analysis, thrombocytopenia cannot be seen as an independent prognostic marker.
It is important to have a high index of suspicion in a patient with thrombocytopenia and any of the above symptoms.
The anaemia, neutropenia and thrombocytopenia may occur either singly or in combination and there is, by definition, no underlying systemic illness to account for them.
The thrombocytopenia reverses shortly after cessation of the drug.
Nevertheless, the rarity of fetal thrombocytopenia and the inability to perform a study do not mean that we can ignore this problem.
The thrombocytopenia is due to dilution, platelet adhesion to circuit surfaces, and to destruction and removal of damaged platelets by the reticuloendothelial system.
In this setting, once other causes of thrombocytopenia have been excluded, the most important question is: should the fetal platelet count be determined?
Although a slightly low platelet count i s commonly seen, severe thrombocytopenia resulting in haemorrhage is most unusual.
It is important to remember that thrombocytopenia may contraindicate the use of a regional technique.
The prognostic value of the haematological (thrombocytopenia) and biochemical (elevated amniotransferases and bilirubin) parameters in neonatal blood has also been investigated.
The toxicity profile of temozolomide is excellent, with thrombocytopenia being the most common toxicity, severe thrombocytopenia occurring in less than 10% of patients.
This article will review current knowledge of the diagnosis and treatment of affected fetuses in alloimmune thrombocytopenia and in immune thrombocytopenic purpura.
What factors can predict the risk of severe thrombocytopenia in the fetus?
