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One possibility i s that the synovial membrane is gradually repopulated with immature mesenchymal and bone marrow cells with altered properties.
Synovial effusion was evident in the wrists, elbows and knees.
Although a specific cell or tissue is described in the studies, the virus is likely to primarily infect synovial cells.
Such cells can be derived from bone marrow, adipose tissue and synovial fluid.
Cartilage receives nutrients from the synovial fluid, which is synthesised by the fibroblasts in the synovial membrane.
Synovial fluid analysis is essential if infection or an associated crystal arthritis is suspected.
It is also unclear why activation of synovial fibroblasts persists even after anti-inflammatory therapy.
Examination of synovial fluid obtained from the right knee revealed no evidence of inflammation.
Now it is possible, using artificial synovial fluid agents such as hyaluronic acid.
Synovial fluid analysis shows high viscosity with relatively few cells that are mainly mononuclear.
However, the results of blood and synovial fluid cultures should be checked after 48 hours.
In order to increase the sensitivity, the synovial fluid may be inoculated into blood culture bottles.
Stimulation of rheumatoid synovial cell collagenase and prostaglandin production by partially purified lymphocyte activating factor (interleukin 1).
However, the disease may affect any synovial joint.
Apatite crystals can be demonstrated in the synovial aspirate.
