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Although in the experiments, slender, intermediate and stumpy cells were counted, we choose to include intermediate cells in the stumpy class.
And increasing slender to stumpy transition period causes behaviour similar to monomorphic trypanosome strains.
In both cases, an increased transition period (dotted lines) prevents oscillations and delays the occurrence of stumpy cells.
It predicts many facets of slender to stumpy transformation that are testable.
Values for the morphometric parameters indicated a position between the slender and the stumpy forms.
At least 250 trypanosomes were examined and classified as slender, dividing, intermediate or stumpy forms.
The only major difference is that when a2=0, stumpy cells appear a little later in the simulation.
One subpopulation that differentiated at the background rate into intermediate cells but never fully developed into stumpy cells.
This is necessary because we observe stumpy cells at all times, in particular, during the exponential growth phase.
We predict that stumpy cells die at a rate of approximately 0.012/h (parameter a4).
Instantaneous transition (dashed lines) causes a drop in peak parasitaemia and earlier occurrence of stumpy cells.
Parameter a4, the stumpy cell death rate, is only marginally significant in mouse 3k.
Slender cells divide rapidly in the bloodstream and other organs before transforming into non-dividing stumpy forms.
Peak parasitaemia is increased and the peak is delayed for both slender and stumpy cells.
Stumpy cells are infectious to the tsetse.
