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These products will enter the maternal circulation through the uterine veins, and may play a role in generating the peripheral syndrome in preeclampsia.
There is no consensus about circulating concentrations of a-tocopherol in preeclampsia relative to normal pregnancy.
There has been increasing interest in clinical trials of antioxidants toward prevention or treatment of preeclampsia.
Since preeclampsia is a complex disorder specific to humans, no single model has fully replicated this condition.
Increased placental apoptosis in pregnancies complicated by preeclampsia.
Prevalence of small low-density lipoproteins is increased in preeclampsia.
In preeclampsia the glomerular barrier is certainly altered and there is an increased excretion of protein including albumin.
Therefore, understanding placentation and the unique primate features may be key to understanding preeclampsia.
Thus, inflammation and oxidative stress might act synergistically to induce vascular dysfunction in preeclampsia.
When these physiologic events are superimposed on a specific prepregnancy phenotype, preeclampsia results.
Echocardiographic left ventricular mass to differentiate chronic hypertension from preeclampsia during pregnancy.
They concluded therefore that this was a useful screening tool for pregnancy induced hypertension and preeclampsia.
We hypothesized that, prior to a first pregnancy, there is a relationship of heightened platelet activation to low plasma volume, both findings observed in preeclampsia.
The incidence of preeclampsia is between 5 and 10% of pregnancies and there is no evidence that this has changed appreciably during the last century.
These are the classic findings of the renal lesion of preeclampsia.
