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Very rapid, active transepithelial fluid absorption occurs in kidney proximal tubule, where the majority of fluid filtered by the glomerulus is reabsorbed.
The glomerular lesions and disappearance of glomeruli compromised the renal function of these animals, a fact reflected in raised serum potassium and urea concentrations.
The growth and maturation of human glomeruli and proximal convolutions from term to adulthood.
Large terminals that take par t in glomeruli also occasionally were seen.
The renal glomeruli showed mesangial hypercellularity with occasional interstitial polymorphs.
Subendothelial fibrinoid deposits were present as was mesangial cell interposition, but not in every affected glomerulus.
F1 terminals within glomeruli are thought to originate from interneuron axons.
This article includes an overview of the early work on glucose transporters in the glomerulus.
Typically, these triads are embedded in more complex synaptic zones known as glomeruli.
A normal glomerulus is photographed on the left.
However, it is prudent to first provide a basic review of the glomerulus in proteinuric forms of kidney disease.
Both size and charge selectivity govern the passage of solutes through the glomeruli.
By 9 weeks of age the glomeruli are scarred as shown above.
The protein filtered by the glomerulus is reabsorbed, through proximal tubular epithelial cells by endocytosis and lysosomal degradation.
Strikingly, glomeruli and primitive-appearing tubules had formed; these appeared to connect up to the polycarbonate membrane.
