They also suggest that neuromuscular hyperexcitability may contribute to elimination.
This is in contrast to nonbenzimidazole anthelminthics which act on the parasite neuromuscular pathways and paralyse them.
This implies a high degree of complexity in the regulation of neuromuscular activities in plant-parasitic nematodes.
The most developed method involves the use of neuromuscular signals to control devices that correspond to movement of the muscles.
The neuromuscular excitability depends on resting membrane and threshold potentials.
Currents through during transmitter discharge from the neuromuscular the fusion pore that forms during exocytosis of a secreting junction.
Some patients may be weak because of the combined effects of cachexia and immobility, without there being primary neuromuscular involvement.
Unfortunately, there is no information on the neuromuscular system of octomacrids.
