In such a case, hardwiring the neuronal machinery in a task-specific way may be the superior adaptation strategy compared with an experiencedependent neuronal plasticity.
Thus, astrocytes release soluble factors that modulate interneuron maturation and the formation of inhibitory contacts in the absence of either neuronal activity or neurotrophin signaling.
Immunocytochemistry and electrophysiology were used to identify protein-expression profiles and to examine functional properties of neuronal and astrocytic phenotypes.
This recruitment of activated mononuclear phagocytes would presumably bring more neurotoxins into the immediate vicinity of neurons, potentially contributing to neuronal injury.
If neuronal migration is disrupted, an abnormality in cell position results.
During nervous system development many neurons are destroyed through apoptosis as a means of reducing neuronal cell numbers.
Identification of regions involved in the binding of alpha-bungarotoxin to the human alpha7 neuronal nicotinic acetylcholine receptor using synthetic peptides.
By exchanging the neuronal medium every 2-3 days, slices survived for a couple of weeks in these conditions.
