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Fibrin is liberated, and thromboses occur, especially in the liver.
The blood plasma, when taken from the vessels, clots or passes from a fluid into a gelatinous or semi-solid condition, which is due to the formation within it of a network of fine threads termed fibrin.
The raw surface is first covered by a layer of coagulated blood and fibrin.
The serum is almost entirely free from corpuscles, these being entangled in the fibrin.
When the thrombus is formed slowly, it consists of white blood cells with a small proportion of fibrin, and, being deposited in successive layers, has a distinctly laminated appearance on section.
Ximelagatran acts by binding directly to free and clot-bound thrombin, thereby preventing the conversion of fibrinogen to fibrin, the primary component of blood clots (28).
Different kinds of derivatives of fibrinogen or fibrin, and concentrations of aprotinin in the plasma, were measured in these samples by immuno essays.
This patient suffered from recurrent pericardial effusions and the susceptibility differences may be caused by various layers of deposits of fibrin.
The final effector molecule of the fibrinolytic system is plasmin, which cleaves fibrin into soluble degradation products.
Other indicators of coagulopathy include consumption of clotting factors, increases in clotting times, increases in levels of fibrin degradation products, and thrombocytopaenia.
It is possible to manipulate the snare underneath chronic foreign bodies that have become encased by fibrin.
The two main components of coagulation are platelets and fibrin.
The placental pathology in the 15 animals examined included congestion of villi, old and fresh infarcts, syncytiotrophoblast degeneration and fibrin deposition.
Isolated seminiferous tubule subjected to reverse fibrin zymography using human urokinase and human plasminogen in the overlay mixture.
The vegetations were sterile, and consisted of fibrin platelet thrombus.
Histology reveals typical mushroom-like pseudomembranes, comprising fibrin, mucus, leucocytes and cellular debris.
These proteolytic activities are metal-dependent and target gelatin, fibrin, fibrinogen and fibronectin, but not collagen or laminin.
Histology of the explanted grafts revealed mild changes consistent with vascular rejection-thrombus formation, fibrin plugging, edema and hemorrhage.
Histologically, the scars consisted of an amorphous substance surrounded by fibrin and host necrotic tissues.
The damaged area in turn predisposes to deposition of fibrin, platelets, and formation of a sterile thrombus, producing nonbacterial thrombotic endocarditis.
