We hypothesize that early adverse experiences result in an increased sensitivity to the effects of stress later in life and render an individual vulnerable to stress-related psychiatric disorders.
Observations among male baboons indicate that higher rank position goes together with protective physiological profiles for stress-related illnesses connected with lower levels of basal cortisol and faster cortisol normalization.
Any segmental phenomena will be handled by constraints that are ranked among the various alignment and stress-related constraints we have just seen.
Many of the biological alterations associated with early stress in preclinical studies have been reported in adults with depression and other stress-related disorders.
The osmobiotic state of the nematodes was associated with the accumulation of a significant amount of stress-related proteins.
Figure 3 illustrates a putative two-step model for oxidative stress-related seed ageing.
Recently, a number of studies in adults have shown that this neuroendocrine axis may be hyporesponsive in a number of stress-related states.
Furthermore, such patients may have more reactive autonomic or stress-related hormonal systems at the outset.