The external urethral sphincter (sphincter urethrae) is under somatic control and is consciously relaxed during micturition.
Brain centers that regulate urination include the pontine micturition center, periaqueductal gray, and the cerebral cortex.
The eggs are eliminated from the host into the water supply with micturition.
In other words, the overall effect of sympathetic stimuli on the bladder is relaxation, in order to delay micturition during stress.
Once the voluntary signal to begin voiding has been issued, neurons in pontine micturition center fire maximally, causing excitation of sacral preganglionic neurons.
During micturition, parasympathetic stimulation causes the detrusor muscle to contract and the internal urethral sphincter to relax.
When stimulating the anterior part, he could induce fall of blood pressure, slowing of respiration and responses such as hunger, thirst, micturition (urination) and defecation.
In the most severe cases, bladder hyperreflexia is associated with a lack of concern for micturition due to the severe frontal lobe cognitive impairment.
