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Effects of brimonidine on aqueous humor dynamics in human eyes.
The source of proteins in the aqueous humor of the normal rabbit.
Effect of clonidine on aqueous humor flow in normal human eyes.
Effects on aqueous humor dynamics in monkeys.
Medications that lower intraocular pressure work by decreasing aqueous humor production and/or increasing aqueous humor outflow.
This vasoconstriction may explain the acute reduction in aqueous humor flow.
Because the ciliary body produces aqueous humor, it is the main target of many medications against glaucoma.
Its inhibition leads to the lowering of aqueous humor production and causes a subsequent drop in the intraocular pressure.
This effect is thought to be caused by reducing the production of the liquid (which is called the aqueous humor) within the eye.
The medications within the eyedrops can include beta blockers (such as levobunolol or timolol) which slow the production of the aqueous humor.
Physostigmine also increases outflow of the aqueous humor in the eye, making it useful in the treatment of glaucoma.
In a healthy cornea, endothelial cells keeps the tissue from excess fluid absorption, pumping it back into the aqueous humor.
Both chemicals work by decreasing the production of aqueous humor.
This accumulation of aqueous humor causes an acute increase of pressure and pain.
It acts via decreasing synthesis of aqueous humor, and increasing the amount that drains from the eye through uveoscleral outflow.
